Chapter

Gene–environment interaction in coronary artery disease: apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples

S. E. Humphries

in Coronary Heart Disease Epidemiology

Second edition

Published in print June 2005 | ISBN: 9780198525738
Published online September 2009 | e-ISBN: 9780191724114 | DOI: https://dx.doi.org/10.1093/acprof:oso/9780198525738.003.0032
Gene–environment interaction in coronary artery disease: apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples

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This chapter discusses gene-environment interaction in coronary artery disease (CAD). Topics covered include risk factors for CAD, apolipoprotein E and effect on lipid levels and coronary heart disease risk, APOE genotype-smoking interaction, homeostasis and use of stressing the genotype to identify functional variants, and the role of inflammatory processes in atherogenesis.

Keywords: coronary artery disease; coronary heart disease; lipid levels; inflammation; candidate genes

Chapter.  8053 words.  Illustrated.

Subjects: Public Health and Epidemiology

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