Related Overviews


'AKT1' can also refer to...


Role of Akt1 in Cardiac Fibrosis Induced by Angiotensin II

Positive association of AKT1 haplotype to Japanese methamphetamine use disorder

Yin Yang 1 promotes mTORC2-mediated AKT phosphorylation

AKT1 and TRH1 are required during root hair elongation in Arabidopsis

Cognitive impairment following prenatal immune challenge in mice correlates with prefrontal cortical AKT1 deficiency

Regulation of IGF-1/PI3K/Akt signalling by the phosphoinositide phosphatase pharbin

Akt1-Mediated Regulation of Macrophage Polarization in a Murine Model of Staphylococcus aureus Pulmonary Infection

AIB1 Promotes DNA Replication by JNK Repression and AKT Activation during Cellular Stress

AKT down-regulates insulin-like growth factor-1 receptor as a negative feedback

Regulation of AKT1 expression by beta-catenin/Tcf/Lef signaling in colorectal cancer cells

TRPV1-antagonist AMG9810 promotes mouse skin tumorigenesis through EGFR/Akt signaling

Physiological activation of Akt by PHLPP1 deletion protects against pathological hypertrophy

Akt activation by PHLPP1 ablation prevents pathological hypertrophy by promoting angiogenesis

Cbl and Akt regulate CXCL8-induced and CXCR1- and CXCR2-mediated chemotaxis

Functional and direct interaction between the RNA binding protein HuD and active Akt1

Overexpression of the rice AKT1 potassium channel affects potassium nutrition and rice drought tolerance

Oncogenic PI3K mutations are as common as AKT1 and SMO mutations in meningioma

Akt- or MEK-mediated mTOR inhibition suppresses Nf1 optic glioma growth


More Like This

Show all results sharing this subject:

  • Chemistry


Show Summary Details

Quick Reference

A protein that serves as an inward rectifier channel for K+ ions in root cells of Arabidopsis thaliana. It is a member of the shaker superfamily of voltage‐gated ion channels ...

Subjects: Chemistry.

Reference entries

Users without a subscription are not able to see the full content. Please, subscribe or login to access all content.