Peptide dependency of alloreactive CD4+ T cell responses
Degeneracy and additional alloreactivity of drug-specific human αβ+ T cell clones
Definition of agonists and design of antagonists for alloreactive T cell clones using synthetic peptide libraries
Modulation of T cell homeostasis and alloreactivity under continuous FTY720 exposure
MO023MALIGNANCIES CONTRIBUTE TO PRESENSITIZATION WITH ALLOREACTIVE T-CELLS PRIOR TO TRANSPLANTATION
Specific suppression of T helper alloreactivity by allo-MHC class I-restricted CD8+CD28- T cells.
Control of TCR V alpha-mediated positive repertoire selection and alloreactivity by differential J alpha usage and CDR3 alpha composition.
Alloantigen specific CD8+CD28− FOXP3+ T suppressor cells induce ILT3+ ILT4+ tolerogenic endothelial cells, inhibiting alloreactivity
SP746NEPHRECTOMY OF THE FAILED FIRST KIDNEY ALLOGRAFT PREDISPOSES KIDNEY TRANSPLANT RECIPIENTS TO PRESENSITIZATION WITH ALLOREACTIVE T-CELLS AND INFERIOR ALLOGRAFT SURVIVAL AFTER RETRANSPLANTATION
Generation of Cytomegalovirus (CMV)—Specific CD4 T Cell Lines Devoid of Alloreactivity, by Use of a Mixture of CMV—Phosphoprotein 65 Peptides for Reconstitution of the T Helper Repertoire
The peptide-specific alloreactive human T cell repertoire varies largely between individuals and is not extended in HLA-A*0205–anti-HLA-A*0201 pairings
Alloreactivity and anti-tumor activity segregate within two distinct subsets of cytokine-induced killer (CIK) cells: implications for their infusion across major HLA barriers
Indoleamine 2,3-dioxygenase increases p53 levels in alloreactive human T cells, and both indoleamine 2,3-dioxygenase and p53 suppress glucose uptake, glycolysis and proliferation
SP720STRATIFICATION OF KIDNEY TRANSPLANTAT RECIPIENTS INTO THOSE WITH LOW AND HIGH ALLOREACTIVE T-CELLS MAY ALLOW SAFE CONVERSION TO mTOR-INHIBITOR-BASED IMMUNOSUPPRESSION
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The T‐cell response to non‐self MHC molecules.
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