APOBEC-1

APOBEC‐1

APOBEC3B Deletion and Risk of HIV-1 Acquisition

APOBEC3 proteins can copackage and comutate HIV-1 genomes

All APOBEC3 family proteins differentially inhibit LINE-1 retrotransposition

The antiretroviral potency of APOBEC1 deaminase from small animal species

Relationship between Human Immunodeficiency Type 1 Infection and Expression of Human APOBEC3G and APOBEC3F

Deaminase-independent inhibition of HIV-1 reverse transcription by APOBEC3G

Sp1 and Sp3 regulate basal transcription of the human APOBEC3G gene

APOBEC-1 dependent cytidine to uridine editing of apolipoprotein B RNA in yeast

HIV-1 Vif binds to APOBEC3G mRNA and inhibits its translation

Analysis of Reptilian APOBEC1 Suggests that RNA Editing May Not Be Its Ancestral Function

Human LINE-1 restriction by APOBEC3C is deaminase independent and mediated by an ORF1p interaction that affects LINE reverse transcriptase activity

Metabolic regulation of ApoB mRNA editing is associated with phosphorylation of APOBEC-1 complementation factor

Quantitative profiling of the full APOBEC3 mRNA repertoire in lymphocytes and tissues: implications for HIV-1 restriction

Intrinsic restriction activity by apolipoprotein B mRNA editing enzyme APOBEC1 against the mobility of autonomous retrotransposons

C→U editing of apolipoprotein B mRNA in marsupials: identification and characterisation of APOBEC–1 from the American opossum Monodelphus domestica

Mutation Vif-22H, which allows HIV-1 to use the APOBEC3G hypermutation to develop resistance, could appear more quickly in certain non-B variants

HIV-1 genome is often defective in PBMCs and rectal tissues after long-term HAART as a result of APOBEC3 editing and correlates with the size of reservoirs