Severe neurological phenotypes of Q129 DRPLA transgenic mice serendipitously created by en masse expansion of CAG repeats in Q76 DRPLA mice
A Unique Origin and Multistep Process for the Generation of Expanded DRPLA Triplet Repeats
Transgenic Mice Harboring a Full-Length Human Mutant DRPLA Gene Exhibit Age-Dependent Intergenerational and Somatic Instabilities of CAG repeats Comparable with Those in DRPLA patients
Single Sperm Analysis of the CAG Repeats in the Gene for Dentatorubral-Pallidoluysian Atrophy (DRPLA): The Instability of the CAG Repeats in the DRPLA Gene is Prominent Among the CAG Repeat Diseases
Protein binding of a DRPLA family through arginine-glutamic acid dipeptide repeats is enhanced by extended polyglutamine
Adenovirus-Mediated Expression of Mutant DRPLA Proteins with Expanded Polyglutamine Stretches in Neuronally Differentiated PC12 Cells. Preferential Intranuclear Aggregate Formation and Apoptosis
Polyglutamine and transcription: gene expression changes shared by DRPLA and Huntington's disease mouse models reveal context-independent effects
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abbr. for dentatorubralpallidoluysian atrophy.
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