SHORT COMMUNICATION: The BN rat strain carries dominant hepatocarcinogen resistance loci
Biochemical Effects of the Mouse Hepatocarcinogen Oxazepam: Similarities to Phenobarbital
Abundance of DNA adducts of methyleugenol, a rodent hepatocarcinogen, in human liver samples
Transcriptomic responses generated by hepatocarcinogens in a battery of liver-based in vitro models
Comparison of Mode of Action of Four Hepatocarcinogens: A Model-Based Approach
Effects of the rodent peroxisome proliferator and hepatocarcinogen, perfluorooctanoic acid, on apoptosis in human hepatoma HepG2 cells
G1-arrested FaO cells re-enter the cell cycle upon stimulation with the rodent non-genotoxic hepatocarcinogen nafenopin
Different patterns of expression of ornithine decarboxylase mRNAs in
rat liver after acute administration of hepatocarcinogens
Different patterns of expression of onithine decarboxylase mRNAs in rat liver after acute administration of hepatocarcinogens
Decreased hepatocyte growth factor level by Wy-14,643, non-genotoxic hepatocarcinogen in F-344 rats.
The rodent non-genotoxic hepatocarcinogen nafenopin suppresses apoptosis preferentially in non-cycling hepatocytes but also elevates CDK4, a cell cycle progression factor.
The non-genotoxic hepatocarcinogen nafenopin suppresses rodent hepatocyte apoptosis induced by TGFbeta1, DNA damage and Fas.
Proinflammatory mesenchymal effects of the non-genotoxic hepatocarcinogen phenobarbital: a novel mechanism of antiapoptosis and tumor promotion
Identification of Specific mRNA Signatures as Fingerprints for Carcinogenesis in Mice Induced by Genotoxic and Nongenotoxic Hepatocarcinogens
Mutagenicity of the potent rat hepatocarcinogen 6BT to the liver of transgenic (lacI) rats: consideration of a reduced mutation assay protocol
Dose-Dependent Indution of GST-P+ Staining Foci by the Rat Hepatocarcinogen Methapyrilene in the Medium-Term Bioassay
Development and Evaluation of a Genomic Signature for the Prediction and Mechanistic Assessment of Nongenotoxic Hepatocarcinogens in the Rat
Hepatic MicroRNA Profiles Offer Predictive and Mechanistic Insights After Exposure to Genotoxic and Epigenetic Hepatocarcinogens
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Any carcinogen that acts specifically or primarily on liver.
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