Overview

L-type channels


'L-type channels' can also refer to...

L-type channels

Single-channel properties of L-type calcium channels from failing human ventricle

A molecular dynamics study of an L-type calcium channel model

Potential of mean force calculations on an L-type calcium channel model

Effect of chloride channel blockers on the cardiac CFTR chloride and L-type calcium currents

Molecular aspects of adrenergic modulation of cardiac L-type Ca2+ channels

Crosstalk between L-type Ca2+ channels and the sarcoplasmic reticulum: alterations during cardiac remodelling

Mechanisms underlying the activation of L-type calcium channels by urocortin in rat ventricular myocytes

L-type calcium channel targeting and local signalling in cardiac myocytes

Chronic potentiation of cardiac L-type Ca2+ channels by pirfenidone

Do local anaesthetics interact with dihydropyridine binding sites on neuronal L-type Ca2+ channels?

Single L-type Ca2+ channel regulation by cGMP-dependent protein kinase type I in adult cardiomyocytes from PKG I transgenic mice

Effects of L‐type Ca2+‐channel blockade, K+ ATP‐channel opening and nitric oxide on human uterine contractility in relation to gestational age and labour

Efficient intracellular multiplication of Legionella pneumophila in human monocytes requires functional host cell L-type calcium channels

L-type voltage-dependent calcium channel α-1C subunit mRNA is present in ejaculated human spermatozoa*

Isolation and characterization of the primary structure of testis-specific L-type calcium channel: implications for contraception.

Changes in L-type calcium channel abundance and function during the transition to pacing-induced congestive heart failure

Increased open probability of single cardiac L-type calcium channels in patients with chronic atrial fibrillation Role of phosphatase 2A

Protein kinase G reverses all isoproterenol induced changes of cardiac single L-type calcium channel gating

 

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A class of voltage-sensitive calcium channels, activated at membrane potentials more positive than −30 mV, found in neurons, neuroendocrine cells, smooth, cardiac, and striated muscle. Their slow inactivation and possible role in long-term potentiation is the basis for the designation.They are inhibited by dihydropyridines, benzodiazepines, and phenylalkylamines but are insensitive to ω-*conotoxin. Mutation can cause hypokalaemic periodic paralysis.

Subjects: Medicine and Health.


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