T-72

T-72

Glomerulonephritis - II (T72-T95)

T. Hale Boggs (1914–72)

Isoform of Vascular Endothelial Cell Growth Inhibitor (VEGI72–251) Increases Interleukin-2 Production by Activation of T Lymphocytes

285. Ashbourne. M. 9 Nov. '72. Mrs. Thrale. T. E. Watson; collation by him 1920.—1788, No. 53.

Letter 72 She is suffering from late hours, in this point Sir T. Peyton submits to his wife, though he is often fault-finding

Interaction of P53 Arg72Pro and MDM2 T309G polymorphisms and their associations with risk of gastric cardia cancer

Accumulation of Vα7.2–Jα33 invariant T cells in human autoimmune inflammatory lesions in the nervous system

The IgV domain of human B7-2 (CD86) is sufficient to co-stimulate T lymphocytes and induce cytokine secretion.

B7–2 (CD86) is essential for the development of IL-4-producing T cells

B7.2-Ig fusion proteins enhance IL-4-dependent differentiation of tumor-sensitized CD8+ cytotoxic T lymphocyte precursors

Prosecutor v Bikindi (Simon), Judgment, Case no ICTR-01-72-T, ICL 709 (ICTR 2008), 2nd December 2008, Trial Chamber III (ICTR)

Oswald T. Avery 1877–1955, Colin MacLeod 1909–72, and Maclyn McCarty 1911–2005

Invariant Vα7.2-Jα33 TCR is expressed in human kidney and brain tumors indicating infiltration by mucosal-associated invariant T (MAIT) cells

CD154–CD40‐independent up‐regulation of B7‐2 on splenic antigen‐presenting cells and efficient T cell priming by staphylococcal enterotoxin A

A self-hsp60 peptide acts as a partial agonist inducing expression of B7-2 on mycobacterial hsp60-specific T cells: a possible mechanism for inhibitory T cell regulation of adjuvant arthritis?

Protection against generalized autoimmunity of the nervous system (GANS), a novel animal model with combined features of EAE, EAN and EAU by a recombinant HIV-1 Tat37–72 peptide-based multiple T cell epitope vaccine

172G>T variant in the 5′ untranslated region of DNA repair gene RAD51 reduces risk of squamous cell carcinoma of the head and neck and interacts with a P53 codon 72 variant

Combined effects of the p53 codon 72 and p73 G4C14-to-A4T14 polymorphisms on the risk of HPV16-associated oral cancer in never-smokers